WildRoseBeef
Range nerd & bovine enthusiast
It's really interesting to learn about the various influences or infusions that were put into the Angus breed over the time it was first founded in around the mid 18th century. To say that the Angus breed is pure is undoubtedly saying that that person is quite ignorant and unknowledgeable about the breed's history. Of course the simple mention of the word "infusion" or "influence" can generate quite the discussion between and among cattle producers, regardless whether they're Angus producers themselves or not.
Now, the reason I created this post was because the last one I made got deleted because I mistakenly flew off the handle a bit too much and got into an unnecessary fight instead of a civil discussion. I'm creating this post because I want to be much more civil about this and also teach some of you who may not know much about the Angus breed (apart from what I wrote on the breed page seen HERE), particularly about the possible or actual influences that were put into the Angus breed itself.
What encouraged me to start this discussion was this quoted by Kfacres, which I reacted to a bit too harshly:
While we're on the subject of breed influences into Angus cattle, there's a high possibility that Shorthorn blood has also been added to the Angus breed as well. In my breed article above I mention (from reading other breed sites on the Angus breed) that the Angus breed nearly went into extinction due to the sudden increase in popularity of the Shorthorn breed. Shorthorns were reportedly used on commercial Angus cattle to improve them, and that there's the possibility that Shorthorn had already been infused into the Angus breed before the Herd Book was closed. This fact has been denied by many breeders and members of various Angus breed societies. However, that really doesn't exactly explain why the heterosis in an Angus-Shorthorn cross is a bit lower than when crossing Angus with Herefords. Nor does it explain why many characteristics of the Angus breed can be matched (sometimes excelled) by the Shorthorn breed. Just a bit of conspiracy to consider.
Then there's the discussion about how the frame size of Angus cattle was able to increase over a short time period (over 50 years), from the average 900 to 1100 lbs to "monster cows" having mature weights around or over (or even well over) 1800 lbs. There's producers that argue that this was only due to heavy selection for larger birth weights, heavier yearling weights and the like in competition for the increasing popularity of the larger and leaner continental breeds that came to North America in the mid to late 70's, but many others agree that it would take a lot longer than 50 years for a single breed to achieve such a jump in frame sizes. It's possible that's where Holstein and/or Continental influences like Chianina or Limousin or Simmental were at play to help give that extra boost on frame sizes in the Angus breed.
Curly Calf Syndrome
Now I want to return to the quote above and talk about the issue about what used to be known as the Curly Calf Syndrome (now known as Arthrogryposis multiplex). There is certainty, from the various research done, that AM is not caused by an outside breed influences, but is merely a gene deletion (hence a defect) that has started in a particular line of Angus cattle. According to THIS LINK, Curly Calf Syndrome has been proven to have started with the maternal grandsire of GAR Precision 1680 (not GAR Precision 1680 himself, which I initially had thought), which was the bull Rito 9J9 of B156 7T26. However, according Angus Australia (AA) in this link , the true origins of CCS is not really known, but rather that 9J9 was "the deepest in the affected pedigrees that an AM carrier has been confirmed." They also state that "[a] very popular bull in the breed called GAR Precision 1680 [(which would be Rito 9J9)] and his even more popular son, CA Future Direction 5321 have been tested to be carriers of the AM condition." So, the question is how far back this AM genetic defect goes, because obviously Rito 9J9's dam and sire both may or would have had to have the defect themselves, among the other ancestors down the line. Who knows if it started even before GAR Precision 1680 was calved?
What is not disputed is that Arthrogryposis Multiplex started from a gene mutation on a single gene, or rather a deletion in only one particular gene (one that would be too complex to even mention here) that is inherited, and is only controlled by a single pair of genes. In other words, an AM calf only comes about when a bull that is an AM carrier with one copy of these gene is mated with a cow that is an AM carrier herself. There's a 50% chance that these two carriers will pass on these gene to their calves which subsequently become carriers themselves, and a 25% chance that they will produce a AM calf. But there's also a 25% chance that the sire and dam will pass on a normal, non-carrier calf. However genetics never work like that in the real world. According to the AA, reports from producers show that there were lower than expected numbers of dead and deformed calves that were confirmed to have AM, so investigation is underway to find out if there is indeed some other factors at play here that affect the incidence and occurrence of AM in this breed.
So the question as to where the Jersey breed comes into play with this can be answered thus: it doesn't. GAR Precision 1680 lines were highly popular for the calving ease and superior weaning and yearling growth (according to Curly Calf "The Straight Story" ), something which can never be found nor generated in the Jersey breed. However, not really much is known about the ancestry of Rito 9J9 and Precision 1680, and that in itself is being investigated along with the research done to see how far back the AM mutation has gone.
I had accredited the presence of Curly Calf to line-breeding, but that, upon further research, is in itself only a half truth. If curly calf was indeed a result of line-breeding, this disease wouldn't be so wide spread, in a manner of speaking, like it has. Instead it would be contained and the sire and dam traced back and culled out of the herd. Line-breeding is done to make a certain line or breed pure, just like inbreeding is, and is the quickest method to weed out certain genetic mutations, abnormalities or defects in a certain line. It is without doubt that many of the Angus cattle today can be traced back to several ancestors several times over, it shouldn't be used as a way to automatically state that line-breeding is at fault for causing curly calf or any other abnormality that is present in the Angus breed. So I was at fault for stating that.
Now, I hope that clears things up for anybody having any questions or anything of that nature in relation to the post I had made in Farmerboy's "Milking Shorthorn cow" thread which I had hijacked and got locked down (sorry for that, FB). This genetic "stuff," for lack of better terms, is certainly interesting and worth the discussion, researching and writing about.
Now, the reason I created this post was because the last one I made got deleted because I mistakenly flew off the handle a bit too much and got into an unnecessary fight instead of a civil discussion. I'm creating this post because I want to be much more civil about this and also teach some of you who may not know much about the Angus breed (apart from what I wrote on the breed page seen HERE), particularly about the possible or actual influences that were put into the Angus breed itself.
What encouraged me to start this discussion was this quoted by Kfacres, which I reacted to a bit too harshly:
Naturally the possibility is there, since a Jersey-Angus cross certainly can produce quite the quality beef, for sure. Great marbling, nice small size, and good carcass characteristics from this cross (particularly if it's 1/2 Angus and 1/2 Jersey or 3/4 Angus and 1/4 Jersey). However I also have to, unfortunately, disagree with this. The reason for this is because the influence of Jersey into the Angus breed, no doubt adding to the traits mentioned above (calving ease, marbling, more milk, etc.), doesn't add any value to the breed particularly where frame size is concerned, not to mention breeding-back ability (or fertility) and muscling ability. You'd certainly get a lot of funnel-butted Angus cattle with this sort of cross, not to mention with a bit of Holstein/Friesian influence, a fact which is much more widely accepted among Angus producers than the fact about the possible Jersey influence. Holstein would definitely add more frame and more milk, not to mention reduce muscling and create more funnel-butt animals, resulting in more dairy-ish looking Angus cattle which are present in a few different lines of this breed. (Heck it's highly likely that there's some Angus influence into some lines of Holstein-Friesian cattle as well!)I think it would be hard to beat a Jersey for a home milk cow- -small, and if you don't poor the grain to them-- they won't give tons. Breed them Angus- and enjoy that beef... After all, what do you think injected the added milk, calving ease, marbling, and CURLY Calf into the Angus breed?
While we're on the subject of breed influences into Angus cattle, there's a high possibility that Shorthorn blood has also been added to the Angus breed as well. In my breed article above I mention (from reading other breed sites on the Angus breed) that the Angus breed nearly went into extinction due to the sudden increase in popularity of the Shorthorn breed. Shorthorns were reportedly used on commercial Angus cattle to improve them, and that there's the possibility that Shorthorn had already been infused into the Angus breed before the Herd Book was closed. This fact has been denied by many breeders and members of various Angus breed societies. However, that really doesn't exactly explain why the heterosis in an Angus-Shorthorn cross is a bit lower than when crossing Angus with Herefords. Nor does it explain why many characteristics of the Angus breed can be matched (sometimes excelled) by the Shorthorn breed. Just a bit of conspiracy to consider.
Then there's the discussion about how the frame size of Angus cattle was able to increase over a short time period (over 50 years), from the average 900 to 1100 lbs to "monster cows" having mature weights around or over (or even well over) 1800 lbs. There's producers that argue that this was only due to heavy selection for larger birth weights, heavier yearling weights and the like in competition for the increasing popularity of the larger and leaner continental breeds that came to North America in the mid to late 70's, but many others agree that it would take a lot longer than 50 years for a single breed to achieve such a jump in frame sizes. It's possible that's where Holstein and/or Continental influences like Chianina or Limousin or Simmental were at play to help give that extra boost on frame sizes in the Angus breed.
Curly Calf Syndrome
Now I want to return to the quote above and talk about the issue about what used to be known as the Curly Calf Syndrome (now known as Arthrogryposis multiplex). There is certainty, from the various research done, that AM is not caused by an outside breed influences, but is merely a gene deletion (hence a defect) that has started in a particular line of Angus cattle. According to THIS LINK, Curly Calf Syndrome has been proven to have started with the maternal grandsire of GAR Precision 1680 (not GAR Precision 1680 himself, which I initially had thought), which was the bull Rito 9J9 of B156 7T26. However, according Angus Australia (AA) in this link , the true origins of CCS is not really known, but rather that 9J9 was "the deepest in the affected pedigrees that an AM carrier has been confirmed." They also state that "[a] very popular bull in the breed called GAR Precision 1680 [(which would be Rito 9J9)] and his even more popular son, CA Future Direction 5321 have been tested to be carriers of the AM condition." So, the question is how far back this AM genetic defect goes, because obviously Rito 9J9's dam and sire both may or would have had to have the defect themselves, among the other ancestors down the line. Who knows if it started even before GAR Precision 1680 was calved?
What is not disputed is that Arthrogryposis Multiplex started from a gene mutation on a single gene, or rather a deletion in only one particular gene (one that would be too complex to even mention here) that is inherited, and is only controlled by a single pair of genes. In other words, an AM calf only comes about when a bull that is an AM carrier with one copy of these gene is mated with a cow that is an AM carrier herself. There's a 50% chance that these two carriers will pass on these gene to their calves which subsequently become carriers themselves, and a 25% chance that they will produce a AM calf. But there's also a 25% chance that the sire and dam will pass on a normal, non-carrier calf. However genetics never work like that in the real world. According to the AA, reports from producers show that there were lower than expected numbers of dead and deformed calves that were confirmed to have AM, so investigation is underway to find out if there is indeed some other factors at play here that affect the incidence and occurrence of AM in this breed.
So the question as to where the Jersey breed comes into play with this can be answered thus: it doesn't. GAR Precision 1680 lines were highly popular for the calving ease and superior weaning and yearling growth (according to Curly Calf "The Straight Story" ), something which can never be found nor generated in the Jersey breed. However, not really much is known about the ancestry of Rito 9J9 and Precision 1680, and that in itself is being investigated along with the research done to see how far back the AM mutation has gone.
I had accredited the presence of Curly Calf to line-breeding, but that, upon further research, is in itself only a half truth. If curly calf was indeed a result of line-breeding, this disease wouldn't be so wide spread, in a manner of speaking, like it has. Instead it would be contained and the sire and dam traced back and culled out of the herd. Line-breeding is done to make a certain line or breed pure, just like inbreeding is, and is the quickest method to weed out certain genetic mutations, abnormalities or defects in a certain line. It is without doubt that many of the Angus cattle today can be traced back to several ancestors several times over, it shouldn't be used as a way to automatically state that line-breeding is at fault for causing curly calf or any other abnormality that is present in the Angus breed. So I was at fault for stating that.
Now, I hope that clears things up for anybody having any questions or anything of that nature in relation to the post I had made in Farmerboy's "Milking Shorthorn cow" thread which I had hijacked and got locked down (sorry for that, FB). This genetic "stuff," for lack of better terms, is certainly interesting and worth the discussion, researching and writing about.